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Apple Watch Onboard SDK for macOS

Aug 17, 2019 · Current version of Apple Watch SDK is 9.1. iPhone SDK 9.1, Xcode 9.3

Jan 29, 2019 · Current version of Apple Watch SDK is 9.0

Apr 2, 2019 · Current version of Apple Watch SDK is 8.0

May 14, 2019 · Current version of Apple Watch SDK is 7.0

Aug 20, 2018 · Current version of Apple Watch SDK is 6.1

Jul 21, 2019 · Current version of Apple Watch SDK is 6.1

Aug 17, 2019 · Current version of Apple Watch SDK is 5.1

Jul 21, 2019 · Current version of Apple Watch SDK is 4.1

Aug 17, 2019 · Current version of Apple Watch SDK is 3.1

Apr 2, 2019 · Current version of Apple Watch SDK is 2.2

Apr 16, 2019 · Current version of Apple Watch SDK is 2.1

Jul 21, 2019 · Current version of Apple Watch SDK is 2.0

Oct 16, 2018 · Current version of Apple Watch SDK is 1.4

Jul 29, 2019 · Current version of Apple Watch SDK is 1.3

Jul 13, 2019 · Current version of Apple Watch SDK is 1.2

Jul 31, 2019 · Current version of Apple Watch SDK is 1.1

Aug 2, 2019 · Current version of Apple Watch SDK is 1.0

Aug 2, 2019 · Current version of Apple Watch SDK is 1.0

Aug 1, 2019 · Current version of Apple Watch SDK is 1.0

Aug 1, 2019 · Current version of Apple Watch SDK is 1.0

Apr 14, 2019 · Current version of Apple Watch SDK is 1.0

Jul 23, 2019 · Current version of Apple Watch SDK is 1.0

Mar 18, 2019 · Current version of Apple Watch SDK is 1.0

Aug 5, 2019 · Current version of Apple Watch SDK is 1.0

Jul 29, 2019 · Current version of Apple Watch SDK is 1.0

Jul 29, 2019 · Current version of Apple Watch SDK is 1.0

Mar 18, 2019 · Current version of Apple Watch SDK is 1.0

Oct 16, 2018 · Current version of Apple Watch SDK is 1.0

Oct 14, 2018 · Current version of Apple Watch SDK is 1.

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Previous studies have shown that high trait anxiety and cocaine addiction are both associated with deficient oxytocin-mediated reward function. The present study sought to clarify whether these findings are due to an altered oxytocin response in the respective pathologies, as well as the involvement of the striatum in these disorders. Participants received intranasal oxytocin, placebo, or no treatment prior to a behavioral monetary task assessing their choice for a probabilistic monetary reward. Behavior during the task was monitored via eye-tracking. Prefrontal cortex (PFC), orbitofrontal cortex (OFC), amygdala, and striatum were investigated via functional magnetic resonance imaging. Oxytocin reduced anticipatory PFC and OFC activity for the binary decision, whereas cocaine-dependent participants received a stronger response to oxytocin in these regions. Furthermore, intra-striatal oxytocin attenuated the effect of cocaine on anticipatory striatal activity. The present results extend previous findings of an altered oxytocin response in emotional and addiction pathology to include a dysfunctional oxytocin response in the dorsal striatum. This suggests that an oxytocin-related impairment of reward function might be an important factor in the development and maintenance of emotional and addiction disorders.Modulation of inflammatory responses by SDE in severe asthma patients.
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